Hyperprolactinemia
Basics
Description
Hyperprolactinemia is an abnormal elevation in the serum prolactin (PRL) level from either physiologic or pathologic influences of the lactotroph cells of the pituitary gland.
Epidemiology
Prevalence
- Predominant age: reproductive age
- Predominant sex: female (70%) > male (30%)
- More readily detected in females because a slight elevation in PRL causes changes in menstruation and galactorrhea; men present with headache, visual disturbances, and erectile dysfunction (1)
- Adenomas in men are typically larger because of delayed onset of symptoms (1).
Etiology and Pathophysiology
- PRL, which is produced by lactotrophs in the anterior pituitary, is regulated by:
- Inhibitory factors, primarily dopamine, are produced in the hypothalamus and delivered via the hypothalamic-pituitary vessels in the pituitary stalk.
- Stimulatory factors, primarily thyrotropin-releasing hormone (TRH)
- Causes of hyperprolactinemia include the following:
- Physiologic
- Pregnancy due to increased estrogen
- Breastfeeding or nipple stimulation
- Stress, including postoperative state
- Dopamine (D2) blockers: prochlorperazine, metoclopramide
- Dopamine depleters: α-methyldopa, reserpine
- Antidepressants: tricyclic antidepressants (TCAs); paroxetine (an SSRI) causes transient hyperprolactinemia—usually resolves in 7 to 10 days
- Gastric motility drugs: metoclopramide and domperidone
- Verapamil (but no other calcium channel blockers; thought to decrease the hypothalamic synthesis of dopamine)
- Older antipsychotics (category is the most common cause of medication induced): haloperidol, fluphenazine, risperidone (level of elevation with risperidone greater than with other antipsychotics)
- Newer antipsychotics (asenapine, iloperidone, lurasidone) may cause elevation but less than the older antipsychotics (1).
- Pathologic
- Hypothyroidism (due to elevated TRH)
- Chest wall conditions such as herpes zoster, trauma, or postthoracotomy
- PRL-secreting adenoma in the anterior pituitary (microadenoma: <1 cm; macroadenoma: >1 cm)
- Pituitary stalk compression/disruption:
- Craniopharyngioma, Rathke cleft cyst
- Meningioma, astrocytoma
- Metastases
- Head trauma
- Infiltrative/inflammatory disorders
- Diminished PRL clearance (chronic renal failure, cirrhosis, cocaine)
- Physiologic
Genetics
Unknown
Risk Factors
See causes in “Etiology and Pathophysiology” section.
General Prevention
Avoid offending medications.
Commonly Associated Conditions
- Infertility
- Osteoporosis
- Amenorrhea
- Gynecomastia
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Citation
Domino, Frank J., et al., editors. "Hyperprolactinemia." 5-Minute Clinical Consult, 33rd ed., Wolters Kluwer, 2025. Medicine Central, im.unboundmedicine.com/medicine/view/5-Minute-Clinical-Consult/116298/all/Hyperprolactinemia.
Hyperprolactinemia. In: Domino FJF, Baldor RAR, Golding JJ, et al, eds. 5-Minute Clinical Consult. Wolters Kluwer; 2025. https://im.unboundmedicine.com/medicine/view/5-Minute-Clinical-Consult/116298/all/Hyperprolactinemia. Accessed December 18, 2024.
Hyperprolactinemia. (2025). In Domino, F. J., Baldor, R. A., Golding, J., & Stephens, M. B. (Eds.), 5-Minute Clinical Consult (33rd ed.). Wolters Kluwer. https://im.unboundmedicine.com/medicine/view/5-Minute-Clinical-Consult/116298/all/Hyperprolactinemia
Hyperprolactinemia [Internet]. In: Domino FJF, Baldor RAR, Golding JJ, Stephens MBM, editors. 5-Minute Clinical Consult. Wolters Kluwer; 2025. [cited 2024 December 18]. Available from: https://im.unboundmedicine.com/medicine/view/5-Minute-Clinical-Consult/116298/all/Hyperprolactinemia.
* Article titles in AMA citation format should be in sentence-case
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